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In this review, we examine how GSH restrains both executioner-driven cell death pathways, including apoptosis, necroptosis, and pyroptosis, which are mediated by defined signaling cascades and dedicated effector proteins, and metabolism-centered forms of cell death, including ferroptosis and cuproptosis, which arise from overwhelming lipid peroxidation and proteotoxic stress, respectively

Using affinity-purification mass spectrometry, Gordon et al

Calcium and apoptosis: ER-mitochondria Ca2+ transfer in the control of apoptosis

FIGURE 2 These kinetic studies of SmTGR occur with the U597C variant background

The conjugation activity towards these allelochemicals was reduced when suppressing GSTE1 gene expression via RNA interference (RNAi), suggesting a role for GSTE1 in host plant adaptation [97]

Specifically, the macrophage population in DW has undergone significant changes