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Role of ferroptosis in PDAC development The tumor suppressive role of ferroptosis Genetic alterations in PDAC often involve key oncogenes and classical cancer signaling pathways

Acknowledgements We thank Yale School Medicine, Yale West Campus, Systems Biology Institute and Cancer Biology Institute for the instrumentation supports

CrossRef The Role of Polyphenols in Regulating Skeletal Muscle Development and Homeostasis: Molecular Mechanisms and Potential Medical Applications Roberto Mattioli, Daniel Di Risola, Carmen Chiaradia, Ivan Dimauro, Luciana Mosca, Guglielmo Duranti, Roberta Ceci Molecular Nutrition & Food Research.2026;[Epub] CrossRef PDK4 knockdown suppresses osteoclast differentiation in osteoporosis through glycolytic flux suppression and AMPK activation Yanling Huang, Rong Zhou, Rulian Liang, Jiping Shen Bone.2026

Mutations in the HFE gene are the major cause of hereditary hemochromatosis, a genetic disorder characterized by impaired regulation of TfR-mediated iron uptake, leading to excessive iron absorption and iron overload in organs such as the liver, heart, pancreas, and joints, ultimately resulting in tissue damage (Brissot et al., 2018

Trends in Cardiovascular Medicine, 25 (6), 475484

3.1 Overview of targeted signaling pathways of natural products The NPs exhibit therapeutic potential in IPF by modulating key signaling pathways involved in inflammation, myofibroblast activation, oxidative stress, and ECM deposition