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TNF and IL-1 but not IL-18 reduces hippocampus long-term potentiation directly at the synapse

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4.2 The potential significance of targeting HIF-1 in different therapies The association between HIF-1 and TNBC strongly suggests the possibility of novel targeted therapy in combination with chemotherapy, anti-angiogenic therapy, and immunotherapy for TNBC treatment (97, 102)

Subsequently, the heterodimer (HIF-:HIF-) migrates to hypoxic response elements (HREs) of the target gene and activates the transcription of multiple genes involved in multiple cellular pathways (35)