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In vitro mechanistic studies also showed that the lead compound C1-27 acts as a slow-turnover substrate, though the significance of this mechanism is currently unclear

R.PalaniappanD.SubramanianR.PatankarM

The persistence of elevated anti-adenovirus IgG titers, complexity of detecting exogenous p53 expression in tumor specimens, and identification of wild-type p53 tumor status as a predictor of clinical response create ambiguity about whether observed antitumor effects are the result of exogenous TP53 expression or immune response to the adenoviral vector (81, 82)

Hannaway, N

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Cells were exposed to 0.4 mM L-Cysteine (102839, Merck) and/or exposed to hypoxia-induced conditions with 0.1 mM cobalt chloride (C8661, Sigma-Aldrich)