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When excessive LPS is transferred to the systemic circulation and increases plasma LPS levels, it can trigger inflammation, thereby facilitating the onset and progression of sarcopenia (Bindels and Delzenne, 2013)

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doi: 10.1007/s12038-019-9973-4

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Conversely, an overproduction of ROS or a deficit in defense mechanisms can lead to the appearance of stress which causes non-specific and irreversible oxidation of biological molecules, engendering dysfunction [1, 4]