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These gaps highlight the need for future research to move beyond descriptive mechanisms toward systematic dissection of pathway crosstalk, cell type-specific functions, and clinical validation

Although these models are instrumental for mechanistic studies, they fail to fully recapitulate the multifactorial and long-term nature of human ovarian aging, which is shaped by decades of environmental exposures, metabolic comorbidities, genetic heterogeneity, and chronic inflamm-aging settings (Wang et al., 2025

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Unlike traditional anti-inflammatories that only suppress symptoms, these supplements for swelling actively promote healing by targeting specific biological pathways

NAFLD iPSC-Heps exhibited higher endogenous FAO compared to controls (difference of 2772 pmol O 2 /min/10 6 cells, p =0.006)

During aging, neutrophils display an extended lifespan and abnormal phenotypic features such as hypersegmentation in secondary lymphoid organs and bone marrow