The pathophysiological mechanisms underlying elevated GGT levels among patients with COVID-19 remain poorly characterized, but potential contributors may include direct viral infection of hepatobiliary tissue, the hosts inflammatory response to the viral infection, drug-induced liver injury, and pre-existing liver disease in a poorly defined proportion of patients [84]
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Simultaneously, the accumulation of lactate can upregulate the expression of inflammation-associated genes through the activation of the HIF-1 signaling pathway (143145)
Group matching with the FEP cohort for age, sex, and parental education was maintained
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