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20 The detection of polyclonal antibodies in lupus sera in comparison to monoclonal antibodies in the case of B-CLL and clinically silent individuals, indicates that the lower yield is not due to possible differences in affinity binding in polyclonal antibodies

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demonstrated that GSH regulates elevation of mitochondrial transmembrane potential ( m ) or mitochondrial hyperpolarization (MHP), which in turn activates mTOR in lupus T cells [91]

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541 It also modestly enhances the skeletal muscle insulin response, increasing glucose uptake