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Inhibition of GPx1 activity partially normalized the cardiomyopathy in Hsp27 transgenic mice, suggesting that reductive stress induced by excess GSH levels and GPx1 activity contributes to cardiac dysfunction in this model (119)

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Such differential targeting of subcellular organelles subsequently resulted in a cell functional change

[DOI] [PubMed] [Google Scholar] 32.Valero T

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