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SIRT3 is also believed as a protection for cardiomyocytes from oxidative stress-mediated cell damage (38)

Inhibition of fatty acid oxidation by etomoxir impairs NADPH production and increases reactive oxygen species resulting in ATP depletion and cell death in human glioblastoma cells

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On the other hand, IL-1 can feedback-stimulate NADPH oxidase, further exacerbating oxidative stress ( 3.2 Oxidative stress and metabolic dysregulation Mitochondrial dysfunction mediated by the SIRT1/NAD + pathway represents a pivotal mechanism in DN pathogenesis, where oxidative stress emerges as a central driver