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[] Under normal conditions, the basal level of HEY2, along with HADC1, restrains excessive energy metabolism-related gene expression to maintain cardiac homeostasis

Several lines of evidence support this contention: 1) L-Trp binds at the interdomain cleft of the VFT, which is a canonical agonist-binding site for class C GPCRs (Kunishima et al., 2000

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