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Activation of AXL as a preclinical acquired resistance mechanism against Osimertinib treatment in EGFR-mutant non-small cell lung cancer cells

It is also funded by Procter & Gamble

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I think thats why we need a ton of research on this, because I think what were going to find is that theres going to be some common themes of how the underlying metabolism is dysregulated

Mitigation: Always dose post-workout (your muscle uptake of glucose is highest then blood glucose crash is less severe) Have fast-acting carbs nearby for the first several doses (fruit, dextrose) Never inject fasted Start at 20 mcg and assess blood glucose response before titrating Consider running a continuous glucose monitor (CGM) for the first cycle Libre or Dexcom gives you real-time data If you're already running GLP-1s (Reta, Tirz, semaglutide), add IGF-1 LR3 cautiously the appetite suppression from GLP-1s means you may not feel hunger signals that would otherwise warn you about low blood glucose

This central nervous system mechanism means PT-141 addresses the psychological and neurological aspects of desire, not just physical arousal