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Its core mechanism lies in mitochondrial regulation, which specifically involves reducing mitochondrial damage, regulating apoptosis and oxidative stress-related pathways, and transmitting protective signals via humoral factors (176, 177)

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Steady-state kinetic measurements Different concentrations of carnosine (0, 0.19, 0.39, 0.78, 1.56, 3.13, 6.25, 12.5, 25, 50, and 100 M) was used as a substrate for CNMT

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