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2C), time spent in the drug-paired compartments was significantly influenced by MOR treatment ( F (2,60) = 11.21, p 0.05)

The expression of HvGR1 was mainly localized to shoot tissues and only slight expression was observed in roots

Kanzawa T, Zhang L, Xiao L, Germano IM, Kondo Y, Kondo S

93.Oakley, A

Environ Pollut 203:4049

Consequently, deficiencies in any of these processes caused by the loss of ATM could be used as therapeutic targets.1110 Consistent with this notion, ATM-null CLL cells are defective in homologous recombination repair (HRR), a deficiency that can be exploited to induce tumor-specific killing via enhanced requirement for HRR upon PARP-inhibition.11 ATM is also implicated in redox homeostasis.12 Deregulation of redox homeostasis results in oxidative stress and occurs either due to increased reactive oxygen species (ROS) production or reduced antioxidant capacity