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851 Intestinal I/R causes dysfunction of intestinal microcirculation and aggravates intestinal injury

These findings offer a theoretical basis for precision treatment of breast cancer chemotherapy involving GSH

doi: 10.1001/jama.2016.17441

Abstract This article explores the potential link between endocrine-disrupting chemicals (EDCs), neuroinflammation, and the development of autism spectrum disorder (ASD)

94 Finally, mTORC1 can also be activated at the Golgi membrane, where it is recruited by the RAB1A GTPase 91 and subsequently activated by Golgi-localized RHEB