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FIGURE 1 Mechanisms of innate immunity in regulating bone metabolism: shared pathways Across the aforementioned innate immune components (immune cells, complement system), several core pathways are repeatedly involved in bone metabolism regulation, forming a shared regulatory network: 1) NF-B signaling pathway: activated by pro-inflammatory cytokines (TNF-, IL-6) or complement products (C5a), it promotes osteoclast differentiation via upregulating NFATc1

For example, tumor cells with mutations in the epidermal growth factor receptor (EGFR) are more dependent on cysteine, increasing their sensitivity to the inhibition of SLC7A11 and the depletion of GSH, which induces ferroptosis ( Figure 2 ) ( Figure 2 4.2 Inducing ferroptosis increases sensitivity to radiotherapy As the importance of ferroptosis in tumors is increasingly recognized, ferroptosis induction in tumor cells has become a hot topic in scientific research