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It normally exists in a quiescent state and serves as a major storehouse for vitamin A

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This disruption has been shown to be associated with increased neuroinflammatory markers and A deposition in transgenic AD mouse models, suggesting that iron dysregulation driven by copper deficiency may exacerbate AD pathology (76,111)

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doi:10.1146/annurev-nutr-061121-084407 Jarrett H, McNulty H, Hughes CF, et al

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