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The lack of HrpA/HrpB TPS in the gonococcus would explain why the gonococcus remains predominantly confined within a vacuole during cellular infection, unlike the meningococcus, which is able to escape, and why the meningococcus, after reaching the cytosol, induces pyroptotic pathways during cellular infection, a phenomenon not described for the gonococcus

However, gliT protoplasts were unable to grow in the presence of gliotoxin (10 g/ml) (Figure 1A) whereas exogenous gliotoxin had no effect on wild-type growth

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