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Evidence supporting a role of domain interaction in mediating apoE4 biology was obtained from in vitro studies in neuronal N2A cells expressing R61T mutated apoE4, which abolishes the domain interaction

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Compared with those in the control group, 124 DEGs/DEPs corresponded to the same gene in the ART NPs group

The application of antioxidant agents can limit oxidative stress-directed damages in vitro

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