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doi: 10.3389/fcell.2025.1611055 Received 13 April 2025 Accepted 02 June 2025 Published 16 July 2025 Volume 13 - 2025 Edited by Rosa Vona, National Institute of Health (ISS), Italy Reviewed by Sidra Islam, Case Western Reserve University, United States Sajal Sen, Massachusetts Institute of Technology, United States Updates Copyright 2025 Qi, Mu, Hu, Li and Liu

In a colorectal cancer (CRC) lung metastasis model, gut dysbiosis-induced TCA accumulation promoted M-MDSC expansion and immunosuppressive function by enhancing glycolysis and epigenetically stabilizing PD-L1 expression via H3K4 mono-methylation, with potential involvement of the Farnesoid X Receptor (FXR) ( Conversely, targeting BA metabolism offers a promising strategy to reduce MDSC-mediated immunosuppression

Exogenous hydrogen sulfide exerts proliferation/anti-apoptosis/angiogenesis/migration effects via amplifying the activation of NF-kappaB pathway in PLC/PRF/5 hepatoma cells

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However, they also showed that GPIs and GPI-anchored mucins could activate p38 MAPK later than ERK1/2, thus increasing IL-12 synthesis and generating an opposing effect in the regulation of the immune response by promoting a Th1 response (Ropert et al., 2001)