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Impairment in Glutathione peroxidase 4 (GPX4) function, which attenuates cellular redox defense, i s also associated with cartilage extracellular matrix (ECM) damage (Miao et al., 2022) implying targeting ferroptosis as a promising new direction for possible interventions in OA pathogenesis

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1 According to this theory, a chronic, low-level deficiency of a nutrient like EGT might not cause an acute, overt disease but may lead to cellular damage that accelerates the onset of age-related chronic illnesses like cardiovascular diseases (CVDs), cancers, and metabolic syndromes