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Yet, without addressing glutathione stability, even the best intentions falter

In -thalassemia, disruption of these transporters, coupled with low hepcidin levels, leads to an imbalance in iron metabolism, further exacerbating iron accumulation in cardiac tissue (60)

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For most of the carcinogenic NAs, the established mechanisms of action involve metabolic activation primarily via cytochrome 450 enzymes (CYPs), and mainly through -hydroxylation, generating highly reactive, electrophilic intermediates, which can ultimately form covalent DNA adducts