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A unified model integrating these concepts is proposed in Figure 3

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Unrestrained replication fork progression in HRASV12- or cyclin E1-overexpressing cells depends on proteins involved in restarting R-loop-stalled forks The unrestrained replication fork progression induced by PARP inhibition in cells treated with R-loop-inducing drugs is dependent on the MUS81-EME1 endonuclease, the DNA helicase RECQ1, the DNA ligase IV (LIG4), and the transcription elongation factor ELL, among others 14,27

Studies have shown faster recovery from Achilles tendon transection, improved healing after muscle tears, and even some evidence of gut mucosal repair in models of inflammatory bowel disease

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