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Cocaine neurotoxicity and neurological damage stem from several mechanisms, including mitochondrial dysfunction (Poon et al., 2007), neuroinflammation (Moran et al., 2005), and autophagy ( 3.1 Vascular effects of cocaine Cocaine impairs vascular function by causing vasoconstriction, reducing cerebral blood flow, and decreasing tissue oxygenation

Its most popular benefit is transporting fats into the mitochondria of cells

High-circulating gut microbiota-dependent metabolite trimethylamine N-oxide is associated with poor prognosis in pulmonary arterial hypertension

Eclache, J

Migraine production may be caused by impaired mitochondrial oxidative metabolism, which interferes with regular neuronal activity (Stuart and Griffiths, 2012)

The exercise task consisted of performing 40 min of cycling at 65% of VO 2peak , immediately followed by cycling at 85% of VO 2peak until exhaustion