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Tight control through PTMs and transcriptional regulation ensures precision and prevents unnecessary organelle loss

PubMed Central , Human Molecular Genetics , vol

The intestine-specific knockout of Cldn7 initiates the colonic inflammation because of the increase of the paracellular flux of the bacterial products from the intestinal lumen to lamina propria, such as LPS, peptidoglycan and N-formyl-L-methionyl-L-leucyl-L-phenylalanine (Tanaka et al., 2015)