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The observed lesions such as rapture, sloughing off of meninges, and hemorrhage was indicative of primary injury while the inflammatory responses that may cause neuronal loss, axonal injury, and impairment of synaptic plasticity indicate the secondary injury process (Andres et al., 2014)

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Salinomycin, a potent inhibitor of XOD and URAT1, ameliorates hyperuricemic nephropathy by activating NRF2, modulating the gut microbiota, and promoting SCFA production

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In line with these data, we found that HIF2 KO NSCLC cells presented almost 2-fold increased 5mC DNA methylation compared to HIF WT and HIF1 KO cells (Fig

YL: Investigation, Writing original draft