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These effects occur through mechanisms including enhanced angiogenesis, modulation of growth factor expression, and direct cytoprotection of gastric epithelial cells

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Therefore, there is growing interest in exploring strategies to modify ApoE expression or function as a potential therapeutic approach to mitigate mitochondrial dysfunction in HD

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Studies using in vitro and in vivo models of AD have shown that targeting oxidative stress has a positive effect on neural degeneration, inflammation, A1-42 aggregation, p-tau formation, GSH levels, iron overload, mitochondrial function, motor dysfunction and learning and memory (Sripetchwandee et al., 2016