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When the inhibitory status of TSC is induced by growth factors, the activation of mTORC1 augments cellular anabolism by stimulating the biosynthesis and inhibiting autophagy 60,68

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These proteins, primary components of tight junctions, undergo continuous assembly and disassembly and enable rapid adaptation and preservation of the epithelial integrity while simultaneously facilitating the passage of molecules via the paracellular route [23]

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