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dicarbonyls such as 3-deoxyglucosone, methyl-glyoxal and glyoxal) with proteins and, to a lesser extent, nucleic acids

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In contrast, CR-fed females not only had reduced plaques, but had reduced glial activation relative to both CR and DL-fed mice, with substantially lower pro-inflammatory gene expression, suggesting that while caloric reduction alone may attenuate plaque pathology, the reduction in plaque deposition observed in the DL-fed mice may not be mediated by glial cells

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In ALS, HHcy and folate deficiency are associated with disease progression and increased neuroinflammation, and loss of motor neurons [192, 193]

TABLE 1 The multiple effects of growth hormone (GH) and growth hormone receptor (GHR) on mitochondrial biogenesis