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Our findings describe a role for mitochondrial plasticity as a mechanism for acute 5FU tolerance in CRC cells: mitochondrial changes acutely adopted by CRC cells in response to 5FU-based standard-of-care treatment result in increased mitochondrial biogenesis, which provides metabolic support for increased pyrimidine scavenging by UCK2 to resolve nucleotide stress

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Because YC-1 is an sGC activator, it has been suggested that CHAC1 is induced through the sGC-cGMP-PKG signaling pathway

IRE1 siRNA relieves endoplasmic reticulum stress-induced apoptosis and alleviates diabetic peripheral neuropathy in vivo and in vitro

doi: 10.1038/sj.ijir.3901522 28 KassDAChampionHCBeavoJA

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