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These interactions result in the accumulation of lipoacylated proteins and a concomitant reduction in iron-sulfur (Fe-S) cluster proteins, thereby activating a protein-toxicity stress response that culminates in cell death (5)

A changes the redox-status of BBB cells, which in turn changes the functioning of mitochondria and disrupts the barrier function of endothelial cells by affecting tight junction proteins

Its mechanism, mirroring that of the naturally occurring Thymosin Beta 4, is pivotal in regenerating tissues and promoting wound healing (2)

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A liposome is a tiny, microscopic bubble made of phospholipids (the same fats that make up your cell membranes)

INTRODUCTION Oxidative stress is a ubiquitous cellular process implicated in the etiology of many illnesses [1-5]