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The optimal timing strategy varies accordingly

Several factors can deplete hepatic glutathione stores: Chronic alcohol consumption that overwhelms detoxification pathways Acetaminophen overdose or prolonged use of hepatotoxic medications Metabolic dysfunction associated with obesity and insulin resistance Viral hepatitis and chronic inflammatory conditions Age-related decline in antioxidant enzyme production Understanding these mechanisms helps explain why both glutathione and NAC have become subjects of intense clinical research for liver support and repair

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Using such integrative, multi-omic approaches that combine tumour molecular features, metagenomic, microbiome/human metabolomics, proteomics and nutrigenomics, MPE can add novel insights into the tumour pathology and host/microbiome interactions, potentially providing microbiome-modulating strategies for cancer prevention and treatment (65, 66)

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