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A comprehensive evaluation of the available data is crucial to understand these potential risks and benefits

Viruses, including SARS-CoV-2 (the virus responsible for COVID-19) and Epstein-Barr Virus (frequently implicated in ME/CFS), are known to hijack the host's cellular machinery to replicate

Moreover, mitochondrial dysfunction and related oxidative stress can activate the innate branch of the immune system, leading to the release of pro-inflammatory cytokines influencing various depression-related pathophysiological mechanisms: monoaminergic neurotransmission disruption, tryptophan degradation toward neurotoxic catabolites, glutamate-related excitotoxicity, decreased neurotrophic factors and alterations in the hypothalamic-pituitary-adrenal axis [3, 33, 59]

A., Dickson, E

Moreover, autophagy functions as a survival mechanism that allows cells to adapt to environmental changes and contributes to cellular health by the selective removal and recycling of damaged cellular components