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In macrophages, the activation of mGluR5 elevates the level of the nuclear receptor peroxisome proliferator-activated receptor (PPAR) g and promotes a shift from M1 to M2 phenotype, leading to reduced IFN- production [180]

This study showed that inhibiting HMGB1 nucleocytoplasmic translocation pharmacologically and deleting HMGB1 in TECs in mice were effective in preventing IRI-AKI, tubular ferroptosis, and inflammation compared to control groups [48]

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Although exogenous factors, including infection and inhaled pollutants, could directly oxidize GSH, endogenous disease-specific mechanisms could also prime the process

This might be associated with the high hydrophilicity of these substances

The beneficial effect of intravenously administered ascorbate has been attributed to its prooxidant properties, which can cause damage to cancer cells