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The neurotoxic effects of AS appear to be most severe in the developing brain, as AS can cause cellular changes in the brain

Although AS-IV has strong lipophilicity (theoretically calculated lipid/water partition coefficient of 2.0), it has a high molecular weight, low hydrophilicity, low permeability on the intestinal mucosa and undergoes paracellular transport in the form of passive transport, resulting in low absorption fraction of AS-IV in various target organs or tissues of the body (208,209)

found that G protein-coupled estrogen receptor activates PI3K/AKT/mTOR signaling, inhibiting ferroptosis through SREBP1/SCD-1-mediated lipogenesis [19]

Specifically, it can lead to major birth defects of the brain and spine called neural tube defects (NTD)

Peptides for Targeting Chondrogenic Induction and Cartilage Regeneration in Osteoarthritis, Liao H.J., Chen H.T., Chang C.H., Journal (Cartilage, September 2024, Epub ahead of print)

Oncotarget 6, 3483134845