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Inserm U1053, Bordeaux University, Bordeaux, France Background: Hepatitis D virus (HDV) infection is associated with accelerated progression of liver disease to cirrhosis

Therefore, we hypothesized that the excessive expression of GJs post-APAP exposure in hepsin / mice could be a key factor contributing to the increased severity of liver damage observed following APAP administration

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Biomolecular coronas can redefine surface properties and mask targeting ligands, altering uptake pathways and biodistribution (Salvati et al., 2013)

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