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In response to oxidative stress, NRF2 dissociates from KEAP1, translocates to the nucleus, and activates genes encoding antioxidant enzymes (e.g., superoxide dismutase SOD) and glutathione synthesis enzymes, reducing reactive oxygen species (ROS) and mitigating oxidative damage ( Therapeutic interventions targeting redox imbalance in fibromyalgia Currently, there is a lack of randomized controlled trials (RCTs) directly evaluating redox-modulating interventions targeting the central nervous system (CNS) in fibromyalgia

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UCP2 and UCP3, homologues of the endogenous mitochondrial uncouplers expressed in the heart, also protect against mitochondrial oxidative damage induced with cardiac ischemia-reperfusion by reducing the production of ROS 84

I find that this is a common issue in people with Hashimotos and can affect the absorption of many vital nutrients