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A novel treatment of cystic fibrosis acting on-target: cysteamine plus epigallocatechin gallate for the autophagy-dependent rescue of class II-mutated CFTR

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It would therefore be crucial to re-think the use of deuterated lipids to counterweight lipid, especially PUFA, peroxidation, as their application may seem promising but can hide severe and unpredictable pathologies raised in the recipient host

Increased passage of these metabolites contributes to the development of symptoms of ASD [26]

132,133 FGFR3-TACC3, a recurrent fusion protein, drives tumorigenesis by promoting kinase transphosphorylation and disrupting chromosomal stability

What are the benefits