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In the context of inflammatory skin diseases, oxidative stress can disrupt the skin's hydrolipid barrier, damage keratinocyte DNA, oxidize lipids in the stratum corneum, and induce the production of pro-inflammatory cytokinesinterleukin-1 (IL-1) and interleukin 33 (IL-33)in the dermis, promoting inflammation and exacerbating the disease (120)

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In patients with ME/CFS, research has shown that the Krebs cycle is often impaired, with blockages occurring at specific enzymatic steps

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