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Role of monocyte chemotactic protein-1 and nuclear factor kappa b in the pathogenesis of proliferative diabetic retinopathy

Our recently conducted preclinical studies in the penetrating TBI animal model, demonstrated zero toxic effects for IP-administered NACA at the highest concentration (600 mg/kg), supporting a safer profile for its use as a mitochondrial-targeted neuroprotection compound for TBI (unpublished data)

Public Health 99 (5), 788791

To view a copy of this licence, visit About this article Cite this article Errichetti, E., Zabotti, A

However, CJC-1295 has a longer half-life compared to MK-677

NETs induce Fundc1 phosphorylation of endothelial cells, inhibit mitochondrial autophagy and lead to mitochondrial ROS overproduction and lipid peroxidation, thus inducing endothelial cell ferroptosis and microvascular dysfunction