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Increased NO flux can cause modification on tyrosine residues that induced the ubiquitin-proteasome pathway enhancing skeletal muscle apoptosis and atrophy [126,127]

Reduction of the soluble cyclic GMP vasorelaxing system in the vascular wall of stroke-prone spontaneously hypertensive rats: effect of the alpha1 -receptor blocker doxazosin

pylori cell extracts treated with mBBr

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2.2.3 Coordinated regulation by signaling pathways In periodontitis, aberrant activation of the Hippo-YAP pathway promotes iron accumulation and lipid peroxidation synergistically by upregulating TFRC and ACSL4 while downregulating GPX4, thereby accelerating ferroptosis-induced periodontal tissue damage (33, 34)

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