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Of interest, treatment of aged rats with IGF-I was shown to upregulate eNOS and improve bioavailability of NO[295], and in vitro experiments in cultured endothelial cells demonstrated that IGF-I treatment reduce ROS production and upregulate eNOS[294]

In line with this finding, homozygous mutation of DJ-1 has been linked to increased mitochondrial oxidative stress in human iPSC-derived DA neurons, a feature that was accompanied by accumulation of -synuclein and oxidized form of DA [42]

Description A dual-peptide compound studied across somatotropic, pituitary, and growth hormone secretagogue signaling pathways

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