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10.5897/BMBR2014-0237 95 KaushikD.YadavJ.KaushikP.SacherD.RaniR

However, its different mechanism of action, combined with a favorable safety profile, makes it an option some researchers consider alongside or instead of incretin-based therapies

49 A deficit in the tricarboxylic acid cycle and ETC function under conditions of excessive superoxide production in the brain would be expected to result in a reduced ability to maintain adequate levels of ATP required for normal neuronal and synaptic functioning

Mitochondrial dysfunction associated with increased oxidative stress and alpha-synuclein accumulation in PARK2 iPSC-derived neurons and postmortem brain tissue

These findings suggest that prebiotic-based UA formulations could improve drug stability and support cancer prevention and therapy

Abakumova, et al