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The Thr308 phosphorylation site serves as a pivotal regulatory point through which CaMKK2, as well as 3-phosphoinositide-dependent kinase-1 (PDK1), trigger the activation of Akt/PKB [87, 142]

177 Both in vitro and in vivo studies have demonstrated that epacadostat can reduce tumor growth and promote the proliferation of T cells and NK cells

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Nucleoside analogs such as ACV and GCV inhibit EBV in vitro

These metabolic products supply nutrients to gut cells and stimulate various receptors, crucially influencing intestinal immune responses [76]