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Anti-inflammatory effects: Downregulates pro-inflammatory cytokines (IL-6, TNF-, IL-1) and reduces oxidative stress Gut barrier integrity: Promotes repair of tight junctions, limiting leaky gut and preventing translocation of inflammatory molecules Angiogenesis and collagen production: Enhances growth of new blood vessels and collagen fibers, accelerating tissue repair after injury or chronic inflammation How BPC-157 decreases inflammation 3 Suppresses pro-inflammatory pathways: Inhibits NF-B signaling, reducing the cascade of inflammatory cytokines Enhances anti-inflammatory factors: Upregulates protective growth factors such as VEGF (vascular endothelial growth factor) and TGF- Reduces oxidative stress: Neutralizes reactive oxygen species (ROS), decreasing tissue damage in the inflamed oral mucosa or stomach lining Restores tissue balance: Unlike broad anti-inflammatories (NSAIDs), BPC-157 doesnt shut down inflammation entirely

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Harvard Medical School (2017)

Aspartate aminotransferase (AST), a major hepatocellular transaminase, participates in amino acid catabolism and synthesis and serves as an indicator of amino acid metabolism (30, 31)

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Oxidative cellular dysfunction is caused by highly reactive substances (such as hydrogen peroxide, superoxide, peroxynitrite and hydroxyl radicals) through a variety of mechanisms, such as lipid peroxidation, enzyme inactivation, mitochondrial respiratory chain failure, DNA alteration and/or cell death