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doi: 10.3389/fimmu.2025.1722463 Received 10 October 2025 Revised 03 December 2025 Accepted 12 December 2025 Published 12 January 2026 Volume 16 - 2025 Edited by Gabriella dEttorre, Sapienza University of Rome, Italy Reviewed by Christina K

Although this adaptation initially helps meet the hearts high energy demands, chronic reliance on FA oxidation results in lipotoxicity, mitochondrial stress, and eventually, impaired cardiac function

The mechanism of APAP hepatotoxicity is similar in humans and mice [104]

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