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These active components help reduce abnormal -syn aggregation, block its neurotoxic effects, inhibit the apoptosis of dopaminergic neurons in the substantia nigra, regulate microglial activation, reduce the release of pro-inflammatory factors such as TNF- and IL-1, enhance antioxidant defense systems (e.g., by upregulating the Nrf2/HO-1 pathway) to mitigate oxidative stress damage, and promote neuroregeneration and synaptic repair

NF-B signaling and the NLRP3 inflammasome Chronic inflammation serves as the core driver of declining renal function following AKI and progression to CKD

Haemostatic effects of levothyroxine and selenomethionine in euthyroid patients with Hashimotos thyroiditis

10.1038/s41398-023-02307-7 Transl

SarmaSSinghDSinghPKharePMangalPSinghSet alFinger millet arabinoxylan protects mice from high-fat diet induced lipid derangements, inflammation, endotoxemia and gut bacterial dysbiosis