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Therefore, it is conceivable that HO inhibition in the brain is not desired, although some have speculated that it may be advantageous in premature infants with intracranial bleeding, where a possibility of enhanced local bilirubin formation exists (Drummond and Kappas, 1986)

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10.7554/eLife.02523 84 DohnerH.PratzK

Here, we confirmed that allicin protected the brain from cerebral injury, which could be ascribed to its antiapoptotic and antiinflammatory effects, as well as the regulation of lipid metabolism, using proteomics and metabolomics analysis