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Patients with DKD also exhibit increased mitochondrial division, resulting in mitochondrial fragmentation, uneven mtDNA distribution, reduced expression of enzyme complexes in the ETC, decreased OXPHOS activity, and enhanced glycolysis as a compensatory mechanism [35, 36]

Kitteringham NR, Powell H, Clement YN, Dodd CC, Tettey JN, Pirmohamed M, Smith DA, McLellan LI, Kevin Park B: Hepatocellular response to chemical stress in CD-1 mice: induction of early genes and gamma-glutamylcysteine synthetase

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Med ., 2016, no

Oct 15 2021;10(10)doi:10.3390/cells10102761

Targeting CAF metabolism or disrupting CAF-mediated metabolic crosstalk may enhance immunotherapy responses in NSCLC