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that AFB1, as the parent compound, inhibited rat liver mitochondrial electron transport at the antimycin A-sensitive site (10) and postulated subsequently (5) that the enhanced liver cell necrosis and acute toxicity observed in the low-protein animals could be due to the low rate of metabolism and clearance of AFB1 with a consequent effect on mitochondrial respiration

AML primary blasts and cell lines harbouring c-KIT and FLT3-ITD mutations showed sensitivity to 2-DG treatment, compared with normal haematopoietic cells (112)

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Findings from animal and in vitro research suggest hesperidins positive effects on blood glucose and lipid levels may be related in part to activation of the AMP-activated protein kinase (AMPK) pathway (Jia 2015